MCB
Home Help [Feedback] [For Subscribers] [Archive] [Search] [Contents]
This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowReprints and Permissions
Right arrow Copyright Information
Right arrow Books from ASM Press
Right arrow MicrobeWorld
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Sompayrac, L
Right arrow Articles by Danna, K J
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Sompayrac, L
Right arrow Articles by Danna, K J

 Previous Article  |  Next Article 

Mol Cell Biol. 1984 August; 4(8): 1661-1663

Less than 40% of the simian virus 40 large T-antigen-coding sequence is required for transformation.

L Sompayrac and K J Danna

ABSTRACT

F8dl is a simian virus 40 early-region deletion mutant that lacks the simian virus 40 DNA sequences between 0.168 and 0.424 map units. Despite this large deletion, cloned F8dl DNA transforms Fisher rat F111 cells and BALB/3T3 clone A31 mouse cells as efficiently as does cloned simian virus 40 wild-type DNA. These results indicate that less than 40% of the large T-antigen-coding sequence is required for efficient transformation.

Mol Cell Biol. 1984 August; 4(8): 1661-1663






Home Help [Feedback] [For Subscribers] [Archive] [Search] [Contents]
J. Bacteriol. J. Virol. Eukaryot. Cell
Microbiol. Mol. Biol. Rev. Clin. Vaccine Immunol. All ASM Journals

Copyright © 1984 by the American Society for Microbiology. All rights reserved.