Molecular and Cellular Biology, October 2001, p. 7115-7115, Vol. 21, No. 20
0270-7306/01 DOI:
Copyright © 2001, American Society for Microbiology. All rights reserved.
AUTHOR'S CORRECTION
Mapping of a Serine-Rich Domain Essential for the
Transcriptional, Antiapoptotic, and Transforming Activities of the
v-Rel Oncoprotein
Cailin
Chen,
François
Agnès, and
Céline
Gélinas
Center for Advanced Biotechnology and Medicine, Department of
Biochemistry, and Cancer Institute of New Jersey, University of
Medicine and Dentistry of New Jersey-Robert Wood Johnson Medical
School, Piscataway, New Jersey 08854-5638
Volume 19, no. 1, p. 307-316, 1999. Since publication of this
article, we found anomalies in Table 2 and Fig. 6. A secondary mutation
was found in the JD214-D10 vector listed in Table 2 that is likely to
be responsible for the transformation-defective phenotype observed in
spleen cells. This unintentional mutation occurred during subcloning
into JD214. Similarly, JD214-A10, also listed in Table 2, was found to
contain a frameshift mutation. Investigation also revealed that mutants
A6.7 and A6.7.10 were not tested in spleen transformation assays,
contrary to what was reported in Table 2, and that the protective
activity of mutant A6.7.10 (Fig. 6B) was derived from cell clones
different from those shown in Fig. 6A. In light of these anomalies, we
withdraw the data in Table 2 and Fig. 6 derived from these mutants.
These mistakes do not affect the other figures in the paper and do not
alter the main conclusion that the transcriptional activity of v-Rel correlates with cell transformation and protection from tumor necrosis
factor alpha-induced cell death. We apologize for any confusion that
these errors may have caused.
Molecular and Cellular Biology, October 2001, p. 7115-7115, Vol. 21, No. 20
0270-7306/01 DOI:
Copyright © 2001, American Society for Microbiology. All rights reserved.