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TOR Signaling Is a Determinant of Cell Survival in Response to DNA Damage
Mol. Cell. Biol. Shen et al. 27: 7007

Supplemental material

Files in this Data Supplement:

  • Supplemental file 1 - Table S1 (Enhanced tah mutant sensitivity to camptothecin and rapamycin)
    MS Word document, 37K.
  • Supplemental file 2 - Fig. S1 (Rapamycin enhances the slow S-phase transit of cells exposed to MMS)
    Zipped TIF document, 101K.
  • Supplemental file 3 - Fig. S2 (Mrc1 is required for rapamycin-induced alterations in S-phase tranti of MMS-treated cells)
    Zipped TIF document, 101K.
  • Supplemental file 4 - Fig. S3 (Cycloheximide treatment results in downregulation of Rad53 levels but fails to alter the extent of Rad53 phosphorylation induced by MMS)
    Zipped TIF document, 371K.
  • Supplemental file 5 - Fig. S4 (Rnr2 levels are unaffected by TORC1 signaling)
    Zipped TIF document, 119K.
  • Supplemental file 6 - Fig. S5 (GAL1 promoted RNR1 overexpression)
    Zipped TIF document, 137K.




This Article
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