
TOR Signaling Is a Determinant of Cell Survival in Response to DNA Damage
Mol. Cell. Biol. Shen et al.
27: 7007
Supplemental material
Files in this Data Supplement:
- Supplemental file 1
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Table S1 (Enhanced tah mutant sensitivity to camptothecin and rapamycin)
MS Word document, 37K.
- Supplemental file 2
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Fig. S1 (Rapamycin enhances the slow S-phase transit of cells exposed to MMS)
Zipped TIF document, 101K.
- Supplemental file 3
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Fig. S2 (Mrc1 is required for rapamycin-induced alterations in S-phase tranti of MMS-treated cells)
Zipped TIF document, 101K.
- Supplemental file 4
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Fig. S3 (Cycloheximide treatment results in downregulation of Rad53 levels but fails to alter the extent of Rad53 phosphorylation induced by MMS)
Zipped TIF document, 371K.
- Supplemental file 5
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Fig. S4 (Rnr2 levels are unaffected by TORC1 signaling)
Zipped TIF document, 119K.
- Supplemental file 6
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Fig. S5 (GAL1 promoted RNR1 overexpression)
Zipped TIF document, 137K.