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Articles

p38 Mitogen-Activated Protein Kinase Promotes Cell Survival in Response to DNA Damage but Is Not Required for the G2 DNA Damage Checkpoint in Human Cancer Cells

Mark S. Phong, Robert D. Van Horn, Shuyu Li, Gregory Tucker-Kellogg, Uttam Surana, Xiang S. Ye
Mark S. Phong
1Lilly Singapore Center for Drug Discovery, Pte Ltd. (Eli Lilly & Company), Singapore, Republic of Singapore
5Department of Pharmacology, Yong Loo Lin School of Medicine, National University of Singapore, Singapore, Republic of Singapore
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Robert D. Van Horn
2Cancer Research Division, Lilly Research Labs, Lilly Corporate Center, Eli Lilly & Company, Indianapolis, Indiana 46285
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Shuyu Li
3Translational Science, Lilly Research Labs, Lilly Corporate Center, Eli Lilly & Company, Indianapolis, Indiana 46285
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Gregory Tucker-Kellogg
1Lilly Singapore Center for Drug Discovery, Pte Ltd. (Eli Lilly & Company), Singapore, Republic of Singapore
6Department of Biological Sciences, Faculty of Science, National University of Singapore, Singapore, Republic of Singapore
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Uttam Surana
4Institute of Molecular and Cell Biology, Agency of Science, Technology, and Research, Singapore, Republic of Singapore
5Department of Pharmacology, Yong Loo Lin School of Medicine, National University of Singapore, Singapore, Republic of Singapore
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Xiang S. Ye
2Cancer Research Division, Lilly Research Labs, Lilly Corporate Center, Eli Lilly & Company, Indianapolis, Indiana 46285
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  • For correspondence: ye_xiang_s@lilly.com
DOI: 10.1128/MCB.00949-09
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Article Information

DOI 
https://doi.org/10.1128/MCB.00949-09
PubMed 
20516219

Published By 
American Society for Microbiology Journals
History 
  • Received July 20, 2009
  • Returned for modification August 14, 2009
  • Accepted May 14, 2010
  • Published online July 9, 2010.

Copyright & Usage 
Copyright © 2010 American Society for Microbiology

Article Versions

  • Accepted Manuscript version (June 01, 2010).
  • You are viewing the most recent version of this article.

Author Information

  1. Mark S. Phong1,5,
  2. Robert D. Van Horn2,
  3. Shuyu Li3,
  4. Gregory Tucker-Kellogg1,6,
  5. Uttam Surana4,5 and
  6. Xiang S. Ye2*
  1. 1Lilly Singapore Center for Drug Discovery, Pte Ltd. (Eli Lilly & Company), Singapore, Republic of Singapore
  2. 2Cancer Research Division, Lilly Research Labs, Lilly Corporate Center, Eli Lilly & Company, Indianapolis, Indiana 46285
  3. 3Translational Science, Lilly Research Labs, Lilly Corporate Center, Eli Lilly & Company, Indianapolis, Indiana 46285
  4. 4Institute of Molecular and Cell Biology, Agency of Science, Technology, and Research, Singapore, Republic of Singapore
  5. 5Department of Pharmacology, Yong Loo Lin School of Medicine, National University of Singapore, Singapore, Republic of Singapore
  6. 6Department of Biological Sciences, Faculty of Science, National University of Singapore, Singapore, Republic of Singapore
  • ↵* Corresponding author. Mailing address: Lilly Research Labs, Lilly Corporate Center, DC 0546, Indianapolis, IN 46285. Phone: (317) 277-1467. Fax: (317) 276-6510. E-mail: ye_xiang_s{at}lilly.com
  • † Supplemental material for this article may be found at http://mcb.asm.org/ .

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p38 Mitogen-Activated Protein Kinase Promotes Cell Survival in Response to DNA Damage but Is Not Required for the G2 DNA Damage Checkpoint in Human Cancer Cells
Mark S. Phong, Robert D. Van Horn, Shuyu Li, Gregory Tucker-Kellogg, Uttam Surana, Xiang S. Ye
Molecular and Cellular Biology Jul 2010, 30 (15) 3816-3826; DOI: 10.1128/MCB.00949-09

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p38 Mitogen-Activated Protein Kinase Promotes Cell Survival in Response to DNA Damage but Is Not Required for the G2 DNA Damage Checkpoint in Human Cancer Cells
Mark S. Phong, Robert D. Van Horn, Shuyu Li, Gregory Tucker-Kellogg, Uttam Surana, Xiang S. Ye
Molecular and Cellular Biology Jul 2010, 30 (15) 3816-3826; DOI: 10.1128/MCB.00949-09
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KEYWORDS

DNA damage
p38 Mitogen-Activated Protein Kinases

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