DOI: 10.1128/MCB.00596-20
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PubMed
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History
- Received November 17, 2020
- Returned for modification December 21, 2020
- Accepted January 14, 2021
- Published online March 24, 2021.
Copyright & Usage
Copyright © 2021 American Society for Microbiology. All Rights Reserved.
Article Versions
- Accepted Manuscript version (February 01, 2021).
- You are viewing the most recent version of this article.
Author Information
- Evan R. Delgadoa,
- Hanna L. Ericksonb,
- Junyan Taoa,
- Satdarshan P. Mongaa,
- Andrew W. Duncana and
- Sayeepriyadarshini Anakkb,c
- aDepartment of Pathology, McGowan Institute for Regenerative Medicine, Pittsburgh Liver Research Center, University of Pittsburgh, Pittsburgh, Pennsylvania, USA
- bDepartment of Molecular and Integrative Physiology, University of Illinois at Urbana-Champaign, Urbana, Illinois, USA
- cCancer Center at Illinois, University of Illinois at Urbana-Champaign, Urbana, Illinois, USA
- Address correspondence to Andrew W. Duncan, duncana{at}pitt.edu, or Sayeepriyadarshini Anakk, anakk{at}illinois.edu.
Evan R. Delgado and Hanna L. Erickson are co-first authors. Evan R. Delgado was determined to be the first, as he brought additional mechanistic ideas with YAP and NUAK2 to the manuscript.
Andrew W. Duncan and Sayeepriyadarshini Anakk are co-senior authors.
Citation Delgado ER, Erickson HL, Tao J, Monga SP, Duncan AW, Anakk S. 2021. Scaffolding protein IQGAP1 is dispensable, but its overexpression promotes hepatocellular carcinoma via YAP1 signaling. Mol Cell Biol 41:e00596-20. https://doi.org/10.1128/MCB.00596-20.
